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Autism linked to metal exposure in babies

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Details regarding Autism Spectrum Disorder (ASD) disorders remain unresolved, including their cause. The scientific literature has included numerous publications on autism, some of them later denied , on what the origin of this language and behavior disorder may be. The journal Nature Communications has published a study that establishes a relationship between the toxins and nutrients absorbed by the fetus in the last months of pregnancy and the first three months of life with the incidence of autism.

In this case, researchers at Mount Sinai Hospital in the United States have analyzed evidence found in the teeth of babies, and have determined that children who showed greater nutrient deficiencies and, in turn, presented greater exposure Toxic metals were linked to various developmental problems, intellectual disabilities, and language deficiencies.

More specifically, the study analyzes the teeth of a sample of children with the autism spectrum and compares them with their healthy brothers and sisters. “We found significant differences in the absorption of metals between children with ASD compared to their siblings, but only during certain periods of development, ” explains Manish Arora, director of the study. “The siblings with ASD had a greater absorption of the lead neurotoxin , at the same time that they showed reduced absorption of the essential elements of manganese and zinc , during the last pregnancy and the first months after birth,” he says. An observation that was “predictive of the severity of ASD 8 to 10 years later.”

However, this relationship has only been observed in 1 in 68 children , and it is not clear that the observed correlation implies a cause – consequence relationship. “We need more studies to determine whether the nutrient differences found in the teeth of children with autism are due to their exposure to them or to a genetic difference in how children absorb and process them .”

References:

Arora, M. (2017). Fetal and postnatal metal dysregulation in autism. Nature Communications. D oi : 10.1038 / ncomms15493

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